Vicente E. Torres, M.D http://www.suhagrarx.com/ ., Ph.D., Kaleab Z. Abebe, Ph.D., Arlene B. Chapman, M.D., Robert W. Schrier, M.D., William E. Braun, M.D., Theodore I. Steinman, M.D., Franz T. Winklhofer, M.D., Godela Brosnahan, M.D., Peter G. Czarnecki, M.D., Marie C. Hogan, M.D., Ph.D., Dana C. Miskulin, M.D., Frederic F. Rahbari-Oskoui, M.D., Jared J. Grantham, M.D., Peter C. Harris, Ph.D., Michael F. Flessner, M.D., Ph.D., Charity G. Moore, Ph.D., M.S.P.H., and Ronald D. Perrone, M.D. For the HALT-PKD Trial Investigators: Angiotensin Blockade in Past due Autosomal Dominant Polycystic Kidney Disease Autosomal dominant polycystic kidney disease is characterized by the progressive development of kidney cysts.1 Hypertension develops early in individuals with ADPKD and is usually associated with the progression of disease.8,9 On the basis of these data, the usage of ACE inhibitors as first-line agents to treat hypertension in individuals with ADPKD has become standard clinical practice, although no randomized, clinical trials of adequate size and quality show their superiority over other antihypertensive agents.10-14 The Halt Progression of Polycystic Kidney Disease trials were made to have sufficient power to ascertain the result of intensive blockade of the RAAS and blood-pressure control on the progression of kidney disease in persons with an early15 or moderately advanced stage of ADPKD.
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